Toxicity in a variety of organs has been reported, including the cardiovascular, respiratory, and central nervous systems [5]. The pulmonary sequelae that result from cocaine use will be reviewed here. Other aspects of cocaine use are discussed separately.

Chronic cocaine use promotes persistent inflammation and structural changes, leading to long-term respiratory impairment. This chronic irritation can result in pulmonary fibrosis (scar tissue formation), which decreases the elasticity and capacity of the lungs to exchange oxygen.

INTRODUCTION: Crack lung is a clinical syndrome of pulmonary toxicity caused by smoking crack, which is a highly potent, addictive, crystalline form of cocaine, within 48 hours of presentation. The clinical presentation can mimic pneumonia, pulmonary embolism, and computed tomography (CT) findings can vary depending on lung involvement. We present a case of crack lung in a young male with ...

Cocaine use can precipitate several acute, severe respiratory syndromes that often require emergency medical intervention. One recognized syndrome is “Crack Lung,” characterized by acute, diffuse alveolar damage that typically appears within 48 hours of smoking the drug.

Cocaine has multiple effects on the lungs, both acute and chronic. Effects of cocaine to the lungs depend on the route of administration (oral, nasal, intravenous), dose size, frequency of exposure, and presence of associated substances like heroin, talc or marijuana.

Cocaine has multiple effects on the lungs, both acute and chronic. Effects of cocaine to the lungs depend on the route of administration (oral, nasal, intravenous), dose size, frequency of exposure, and presence of associated substances like heroin, talc or marijuana.

Key findings include the high prevalence of chronic obstructive pulmonary disease among heroin users, the association of cocaine with acute and chronic pulmonary conditions, and the role of alcohol in exacerbating respiratory infections and acute respiratory distress syndrome.

Early recognition of cocaine-associated acute lung injury is crucial to initiate appropriate supportive care and reduce the risk of recurrent respiratory failure.

Repeated exposure to toxic chemicals and irritants in cocaine smoke can cause scarring of the lung tissue, a condition known as pulmonary fibrosis. In pulmonary fibrosis, the lungs lose their elasticity, making it difficult for the alveoli to expand and contract during breathing.

The inhalation of CC exposes the lungs to various toxic compounds, resulting in acute and chronic pulmonary injury. Several mechanisms contribute to that damage.